Review



rabbit anti rat antibodies against col iii  (Proteintech)


Bioz Verified Symbol Proteintech is a verified supplier  
  • Logo
  • About
  • News
  • Press Release
  • Team
  • Advisors
  • Partners
  • Contact
  • Bioz Stars
  • Bioz vStars
    • 96
      Buy from Supplier

    Structured Review

    Proteintech rabbit anti rat antibodies against col iii
    RCDs/UA@Lipo alleviated H 2 O 2 -induced oxidative stress and TGF-β1 induced fibrosis-specific markers separately. ( A ) Immunofluorescence staining images of intracellular ROS and superoxide detected by DCFH-DA probe. ( B ) mitochondrial membrane potential (ΔΨm) detected by JC-1 probe. ( C ) tendon fibrosis markers <t>(COL</t> I, COL <t>III,</t> α-SMA) of NIH3T3. ( D–H ) Semiquantitative analysis of the relative fluorescent intensity of ( A–C ) (n = 3). ( D and E ) Model group is H 2 O 2 group, ( F–H ) Model group is TGF-β1 group. Data are presented as mean ± SD; comparisons between the groups were performed one-way ANOVA. *P < 0.05; **P < 0.01; ***P < 0.001.
    Rabbit Anti Rat Antibodies Against Col Iii, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 2370 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit anti rat antibodies against col iii/product/Proteintech
    Average 96 stars, based on 2370 article reviews
    rabbit anti rat antibodies against col iii - by Bioz Stars, 2026-03
    96/100 stars

    Images

    1) Product Images from "Antioxidant Carbon Dots and Ursolic Acid Co-Encapsulated Liposomes Composite Hydrogel for Alleviating Adhesion Formation and Enhancing Tendon Healing in Tendon Injury"

    Article Title: Antioxidant Carbon Dots and Ursolic Acid Co-Encapsulated Liposomes Composite Hydrogel for Alleviating Adhesion Formation and Enhancing Tendon Healing in Tendon Injury

    Journal: International Journal of Nanomedicine

    doi: 10.2147/IJN.S466312

    RCDs/UA@Lipo alleviated H 2 O 2 -induced oxidative stress and TGF-β1 induced fibrosis-specific markers separately. ( A ) Immunofluorescence staining images of intracellular ROS and superoxide detected by DCFH-DA probe. ( B ) mitochondrial membrane potential (ΔΨm) detected by JC-1 probe. ( C ) tendon fibrosis markers (COL I, COL III, α-SMA) of NIH3T3. ( D–H ) Semiquantitative analysis of the relative fluorescent intensity of ( A–C ) (n = 3). ( D and E ) Model group is H 2 O 2 group, ( F–H ) Model group is TGF-β1 group. Data are presented as mean ± SD; comparisons between the groups were performed one-way ANOVA. *P < 0.05; **P < 0.01; ***P < 0.001.
    Figure Legend Snippet: RCDs/UA@Lipo alleviated H 2 O 2 -induced oxidative stress and TGF-β1 induced fibrosis-specific markers separately. ( A ) Immunofluorescence staining images of intracellular ROS and superoxide detected by DCFH-DA probe. ( B ) mitochondrial membrane potential (ΔΨm) detected by JC-1 probe. ( C ) tendon fibrosis markers (COL I, COL III, α-SMA) of NIH3T3. ( D–H ) Semiquantitative analysis of the relative fluorescent intensity of ( A–C ) (n = 3). ( D and E ) Model group is H 2 O 2 group, ( F–H ) Model group is TGF-β1 group. Data are presented as mean ± SD; comparisons between the groups were performed one-way ANOVA. *P < 0.05; **P < 0.01; ***P < 0.001.

    Techniques Used: Immunofluorescence, Staining, Membrane

    RCDs/UA@Lipo-HAMA reduced adhesion of injured tendons at macroscopic and histological levels at various time points post-injury. ( A ) Schematic diagram of surgical procedures of the ATI. ( B ) Gross view of ATI. ( C )Representative images of immunofluorescence staining of tendon antioxidant (Nrf-2, HO-1) and anti-inflammatory markers (CD68, iNOS) at 2 weeks post-injury (n = 3). ( D ) Representative images of immunofluorescence staining of tendon antifibrosis (COL III, α-SMA) at 6 weeks post-injury (n = 3). ( E ) Representative images of immunohistochemical staining of tendon markers (Vimentin, MMP2, α-SMA) antifibrosis at 6 weeks post-injury (n = 4). ( F–N ) Semiquantitative analysis of expression level of tendon marker of ( C–E ), respectively. Data are presented as mean ± SD; comparisons between the groups were performed by one-way ANOVA. *P < 0.05; **P < 0.01; ***P < 0.001; ****P < 0.0001.
    Figure Legend Snippet: RCDs/UA@Lipo-HAMA reduced adhesion of injured tendons at macroscopic and histological levels at various time points post-injury. ( A ) Schematic diagram of surgical procedures of the ATI. ( B ) Gross view of ATI. ( C )Representative images of immunofluorescence staining of tendon antioxidant (Nrf-2, HO-1) and anti-inflammatory markers (CD68, iNOS) at 2 weeks post-injury (n = 3). ( D ) Representative images of immunofluorescence staining of tendon antifibrosis (COL III, α-SMA) at 6 weeks post-injury (n = 3). ( E ) Representative images of immunohistochemical staining of tendon markers (Vimentin, MMP2, α-SMA) antifibrosis at 6 weeks post-injury (n = 4). ( F–N ) Semiquantitative analysis of expression level of tendon marker of ( C–E ), respectively. Data are presented as mean ± SD; comparisons between the groups were performed by one-way ANOVA. *P < 0.05; **P < 0.01; ***P < 0.001; ****P < 0.0001.

    Techniques Used: Immunofluorescence, Staining, Immunohistochemical staining, Expressing, Marker



    Similar Products

    rabbit anti rat antibodies against col iii  (Proteintech)
    96
    Proteintech rabbit anti rat antibodies against col iii
    RCDs/UA@Lipo alleviated H 2 O 2 -induced oxidative stress and TGF-β1 induced fibrosis-specific markers separately. ( A ) Immunofluorescence staining images of intracellular ROS and superoxide detected by DCFH-DA probe. ( B ) mitochondrial membrane potential (ΔΨm) detected by JC-1 probe. ( C ) tendon fibrosis markers <t>(COL</t> I, COL <t>III,</t> α-SMA) of NIH3T3. ( D–H ) Semiquantitative analysis of the relative fluorescent intensity of ( A–C ) (n = 3). ( D and E ) Model group is H 2 O 2 group, ( F–H ) Model group is TGF-β1 group. Data are presented as mean ± SD; comparisons between the groups were performed one-way ANOVA. *P < 0.05; **P < 0.01; ***P < 0.001.
    Rabbit Anti Rat Antibodies Against Col Iii, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit anti rat antibodies against col iii/product/Proteintech
    Average 96 stars, based on 1 article reviews
    rabbit anti rat antibodies against col iii - by Bioz Stars, 2026-03
    96/100 stars
    		  Buy from Supplier

    Image Search Results


    RCDs/UA@Lipo alleviated H 2 O 2 -induced oxidative stress and TGF-β1 induced fibrosis-specific markers separately. ( A ) Immunofluorescence staining images of intracellular ROS and superoxide detected by DCFH-DA probe. ( B ) mitochondrial membrane potential (ΔΨm) detected by JC-1 probe. ( C ) tendon fibrosis markers (COL I, COL III, α-SMA) of NIH3T3. ( D–H ) Semiquantitative analysis of the relative fluorescent intensity of ( A–C ) (n = 3). ( D and E ) Model group is H 2 O 2 group, ( F–H ) Model group is TGF-β1 group. Data are presented as mean ± SD; comparisons between the groups were performed one-way ANOVA. *P < 0.05; **P < 0.01; ***P < 0.001.

    Journal: International Journal of Nanomedicine

    Article Title: Antioxidant Carbon Dots and Ursolic Acid Co-Encapsulated Liposomes Composite Hydrogel for Alleviating Adhesion Formation and Enhancing Tendon Healing in Tendon Injury

    doi: 10.2147/IJN.S466312

    Figure Lengend Snippet: RCDs/UA@Lipo alleviated H 2 O 2 -induced oxidative stress and TGF-β1 induced fibrosis-specific markers separately. ( A ) Immunofluorescence staining images of intracellular ROS and superoxide detected by DCFH-DA probe. ( B ) mitochondrial membrane potential (ΔΨm) detected by JC-1 probe. ( C ) tendon fibrosis markers (COL I, COL III, α-SMA) of NIH3T3. ( D–H ) Semiquantitative analysis of the relative fluorescent intensity of ( A–C ) (n = 3). ( D and E ) Model group is H 2 O 2 group, ( F–H ) Model group is TGF-β1 group. Data are presented as mean ± SD; comparisons between the groups were performed one-way ANOVA. *P < 0.05; **P < 0.01; ***P < 0.001.

    Article Snippet: The fresh tissue sections were incubated with rabbit anti-rat antibodies against COL III (1:200; Cat#22734-1-AP, Proteintech, USA) and α-SMA (1:200; Cat#19245S, Cell Signaling Technology, USA) at 4°C overnight.

    Techniques: Immunofluorescence, Staining, Membrane

    RCDs/UA@Lipo-HAMA reduced adhesion of injured tendons at macroscopic and histological levels at various time points post-injury. ( A ) Schematic diagram of surgical procedures of the ATI. ( B ) Gross view of ATI. ( C )Representative images of immunofluorescence staining of tendon antioxidant (Nrf-2, HO-1) and anti-inflammatory markers (CD68, iNOS) at 2 weeks post-injury (n = 3). ( D ) Representative images of immunofluorescence staining of tendon antifibrosis (COL III, α-SMA) at 6 weeks post-injury (n = 3). ( E ) Representative images of immunohistochemical staining of tendon markers (Vimentin, MMP2, α-SMA) antifibrosis at 6 weeks post-injury (n = 4). ( F–N ) Semiquantitative analysis of expression level of tendon marker of ( C–E ), respectively. Data are presented as mean ± SD; comparisons between the groups were performed by one-way ANOVA. *P < 0.05; **P < 0.01; ***P < 0.001; ****P < 0.0001.

    Journal: International Journal of Nanomedicine

    Article Title: Antioxidant Carbon Dots and Ursolic Acid Co-Encapsulated Liposomes Composite Hydrogel for Alleviating Adhesion Formation and Enhancing Tendon Healing in Tendon Injury

    doi: 10.2147/IJN.S466312

    Figure Lengend Snippet: RCDs/UA@Lipo-HAMA reduced adhesion of injured tendons at macroscopic and histological levels at various time points post-injury. ( A ) Schematic diagram of surgical procedures of the ATI. ( B ) Gross view of ATI. ( C )Representative images of immunofluorescence staining of tendon antioxidant (Nrf-2, HO-1) and anti-inflammatory markers (CD68, iNOS) at 2 weeks post-injury (n = 3). ( D ) Representative images of immunofluorescence staining of tendon antifibrosis (COL III, α-SMA) at 6 weeks post-injury (n = 3). ( E ) Representative images of immunohistochemical staining of tendon markers (Vimentin, MMP2, α-SMA) antifibrosis at 6 weeks post-injury (n = 4). ( F–N ) Semiquantitative analysis of expression level of tendon marker of ( C–E ), respectively. Data are presented as mean ± SD; comparisons between the groups were performed by one-way ANOVA. *P < 0.05; **P < 0.01; ***P < 0.001; ****P < 0.0001.

    Article Snippet: The fresh tissue sections were incubated with rabbit anti-rat antibodies against COL III (1:200; Cat#22734-1-AP, Proteintech, USA) and α-SMA (1:200; Cat#19245S, Cell Signaling Technology, USA) at 4°C overnight.

    Techniques: Immunofluorescence, Staining, Immunohistochemical staining, Expressing, Marker